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The cancer cells were preincubated with NAC and subsequently treated with NP4 showed a reduced number of TNF- secreted from the macrophages, suggesting that macrophage activation is associated with the NP4-induced oxidative stress

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When overexpressed, ACADSB, a member of the acyl-CoA dehydrogenase, that negatively regulates glutathione reductase and GPX4, was shown to limit CRC cell migration, invasion, and proliferation by promoting CRC cell ferroptosis [151]

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