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While mucosal barrier dysfunction and dysregulated T cell responses collectively drive IBD pathogenesis 5,10 , our assessment of IPAs immunomodulatory effects revealed no significant alterations in colonic lamina propria mononuclear cell (cLPMC) composition, including macrophages, dendritic cells, neutrophils, CD4 + and CD8 + T cell populations at D0 and D7 post-DSS challenge (Supplementary Fig

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McArthur et al., 2013)

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