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2.3 Dysregulation and failure of endogenous antioxidant defense mechanisms Under physiological conditions, the myocardium maintains redox homeostasis through a robust endogenous antioxidant defense system, comprising enzymatic components like SOD, Catalase (CAT), and Glutathione Peroxidases (GPxs), as well as non-enzymatic antioxidants such as glutathione (GSH) and thioredoxin (Kumar et al., 2022)

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At the optimal cut-off value of 12.5 ng/ml, CSE-MS demonstrated 65% sensitivity and 60% specificity, with positive and negative predictive values of 62% and 63%, respectively

Propofol inhibits superoxide production, elastase release, and chemotaxis in formyl peptide-activated human neutrophils by blocking formyl peptide receptor 1