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doi: 10.1016/J.ECOENV.2018.02.057 17 AnderssonM.KarumbunathanV.ZimmermannM

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Since mutant ChaC1-knock-in mice have been examined only in limited situations, it remains ambiguous whether a deficiency of CHAC 1 activity affects phenotypic properties under pathological conditions such as ER stress, oxidative stress, and ferroptosis

Msp1/ATAD1 maintains mitochondrial function by facilitating the degradation of mislocalized tail-anchored proteins

Nowadays consumers are increasingly concerned about the quality and safety of many products of industrialized countries, in particular the use of artificial sweeteners, flavorings, colorings, preservatives and dietary supplements