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Discussion Herein, we demonstrate that obesity-induced insulin resistance is characterized by increased fat cell glutaminolysis, which is evident at the transcript, protein and metabolite levels in both humans and mice

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Use of the composition of claim 1, or the vector system of claim 2 or any claim dependent thereon for genome engineering, provided that said use is not a method for treatment of the human or animal body by surgery or therapy, and provided that said use is not a process for modifying the germline genetic identity of human beings

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This blockage is typically caused by a buildup of plaques caused by atherosclerosis