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The primary reason is that inhaled hydrogen would directly impact the alveoli, Type I and Type II alveolar cells, and would rapidly diffuse into the pulmonary capillaries

In this scenario, it becomes very attractive to study and explore the endothelium, through which inflammatory cells must transmigrate [transendothelial migration (TEM)] to reach the lung parenchyma and cause damage ultimately leading to COPD onset and progression

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