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47 The dysregulated Wnt/-catenin pathway contributes to diabetic kidney disease (DKD)-related renal dysfunction through multifaceted mechanisms: In podocytes, chronic activation induces slit diaphragm protein (nephrin, podocin) downregulation via Snail1-mediated repression and promotes apoptosis through Bim upregulation, exacerbating albuminuria (podocyte-specific -catenin knockout reduces albuminuria by 40% in diabetic models)

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