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Specifically, HMGB1 released from necrotic cells binds and activates RAGEs, TLR-2, and TLR-4 which upregulate the expression of matrix metalloproteinase 9 (MMP-9), tumor necrosis factor- (TNF-), and interleukin (IL)-1, all of which promote cellular death and neuroinflammation [26]

For instance, the astrocytes near the neurons behave as a glucose storage unit and regulate sufficient release of glucose to neurons in case of shortage

Loukas, M., Benninger, B., Tubbs, S.R

Under pathological conditions, oocyte development may be disturbed, resulting in follicular atresia and ovulatory dysfunction [103]

doi: 10.1007/s00294-003-0415-2 32 DenegaI.d'EnfertC.Bachellier-BassiS