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In contrast, inhibiting IKK/NF-B signaling led to a reduction in circulating memory T cells while increasing T RM populations in the lung (169)

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Acid-labile subunit (ALS) and IGFBP-3 are two proteins that bind IGF-I (~90% of total serum IGF-I) in a ternary complex which transports and prolong IGF-I half-life in circulation[23]

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