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Over the last several years, increasing evidence from genetics, neuroimaging, protein expression, and animal model studies supporting the notion of altered glutamate metabolism has heightened the interest in evaluating glutamatergic dysfunction in ASD

The higher level of mitochondrial superoxide in ATM-null CLLs indicated mitochondrial dysfunction

231,232,233 Other studies 234,235 have also reported the protective role of GPX4 in DTP cells against oxidative stress

50 The thiol group acts as a proton donor and is responsible for the biological activity of GSH