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Data are reported as means the standard error of the mean (SEM), representing at least three independent experiments

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Effect of Ang II on medullary RBF Ang II elicits a paradoxical medullary vasodilatation in normotensive animals, mediated by a secondary activation of vasodilator paracrine agents such as prostaglandins, kinins and NO, rather than to a direct action via AT 2 receptors

The theory suggests these peptides may influence gene expression through epigenetic mechanisms, though the precise molecular interactions remain an area of ongoing investigation