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Koga K, Osuga Y, Yoshino O, Hirota Y, Yano T, Tsutsumi O, Taketani Y (2005) Elevated interleukin-16 levels in the peritoneal fluid of women with endometriosis may be a mechanism for inflammatory reactions associated with endometriosis

Our results demonstrated that LPS- exposure induced the inflammatory response, including cytokine expression (IL6, IL-1, TNF) in macrophages, along with Trp-catabolic enzymes IDO1 and associated catabolites Kyn levels in both THP1 and primary KCs

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In particular, a study of the metabolome and of mitochondrial function in a small group of children with 22q11.2DS revealed a distinct biochemical profile, which was consistent with increased oxidative stress, and shared features with congenital propionic and/or methylmalonic acidaemia (6)