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Homozygosity for a partial deletion of apoprotein A-V signal peptide results in intracellular missorting of the protein and chylomicronemia in a breast-fed infant

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Enasidenib also showed clinical activity in patients with higher-risk MDS, both in combination with azacitidine in newly diagnosed patients and as monotherapy in HMA-pretreated patients

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albicans takes the opportunity to multiply excessively, the transition from the yeast phase to the pathogenic mycelial phase, express adhesion molecules, release C