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Description

233 , R67R79 (2017)

This mechanism is highly effective given the rapid kinetics for glutathionylation/deglutathionylation reactions and their depen- dence on mitochondrial bioenergetics

This disease features epithelial injury and abnormal activation of lung fibroblasts, resulting in excessive deposition of extracellular matrix (ECM) proteins, irreversible fibrosis, and destruction of lung architecture