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Only the liver and kidneys can produce NAD + de novo starting from Trp 39

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The NLRP3 inflammasome functions as a driver of the myelodysplastic syndrome phenotype

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Mechanistically, the PR-DUB complex binds on the SLC7A11 promoter, allowing BAP1 to deubiquitinate H2Aub on the SLC7A11 promoter and subsequently repress SLC7A11 expression independent of p53 (Fig