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Recent studies have also reported that DHEA reduces oxidative stress and apoptosis of iron-induced epilepsy via activation of the NRF2/ARE signaling pathway (Pagni and Zenga, 2005)

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Previous reports from our laboratory showed that 15LO1 also contributes to downstream T2 immunoinflammatory responses (6, 10, 14, 28)

Experimental mouse models and human lung organoid models for studying chronic obstructive pulmonary disease

These results suggest a potential regulatory relationship between thymosin beta 4, microRNA 146a expression, and downstream inflammatory pathway modulation