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However, inadequate NADPH production in cells with restricted glucose intake significantly augments actin cytoskeletal disulfide bonds, causing disruption in the actin network and intracellular disulfide hyperplasia

Food Chem Toxicol 50:41034108

Under normal conditions, the released GABA spills over to the neighboring excitatory synaptic terminals and inhibits presynaptic glutamate release through GABA receptors

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But in terms of protein expression, the reduction is not as significant as GPX4