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Cancer 124, 16231636

Knockdown of BCAT1 causes accumulation of -KG, promoting EGLN1-mediated HIF1 protein degradation and leukemia-initiating arrest (Raffel et al., 2017)

Further mechanistic investigations determined that the elevation of intracellular GSSG levels resulted in NF-B inhibition by facilitating glutathionylated IKK- (IKK--SSG) formation, leading to enhanced TNF hepatotoxicity

The use of different moieties has been extensively discussed in other works and goes beyond the scope of the present paper (Blanco et al., 2015)

It has the ability to prevent damage from reactive oxygen species (ROS), free radicals, peroxides, lipid peroxides and heavy metals to cells