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Studies have shown that RIPC can reduce myocardial infarction size and troponin levels by regulating calcium channel subunits, and it can also inhibit the NOX4-ROS signaling pathway to alleviate renal mitochondrial dysfunction, thereby lowering the risk of postoperative renal injury (178, 179)

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Actin remodeling is essential for TNT initiation and elongation, and inhibition of actin polymerization markedly reduces intercellular mitochondrial transport

Cytometry 2:86, 1981

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