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doi: 10.1128/mBio.01139-20 95 MuefongCNSutherlandJS

Bile acids induce mitochondrial ROS, which promote activation of receptor tyrosine kinases and signaling pathways in rat hepatocytes

Staining using an antibody specific to the human TCR variable beta chain 5.1 (TCRV5.1), and an HLA-A*02-S 20 multimer allowed to estimate TCR expression and pairing efficiency, respectively

The nonenzymatic functions of GSTs involve their interactions with cellular proteins, such as, Jun N-terminal kinase,(JNK), tumor necrosis factor receptor-associated factor-2 (TRAF2), apoptosis-signal-regulating kinase 1 (ASK), serine/threonine kinases (PKA, PKC), and tissue transglutaminase 2 (TGM2), during which, either the interacting protein partner undergoes functional alteration or the GST protein itself is post-translationally modified and/or functionally altered [53], [74]

GSOR is then cleaved into GSSG and ROH by a second nucleophilic substitution by the GSH molecule according to a mechanism that is not yet fully elucidated (Deponte, 2013)