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Kim E, Yang J, Park S, Shin K

Using in vitro human models, we showed that these thiomethyl metabolites are directly linked to NAPQI detoxification since they are mainly formed after exposure to glutathione-derived conjugates via an overlooked pathway called the thiomethyl shunt

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however, these medications often turn the acute inflammation and rapid progression of renal disease into chronic inflammation and slow, but do not stop, the still inevitable progression of end-stage renal disease