Pathological angiogenesis can exacerbate neurodegeneration by promoting BBB dysfunction, neuroinflammation, and vascular instability (Sweeney et al

Disclosure of interest: This study was funded by Hong Kong Health and Medical Research Fund E-Posters - RISK FACTORS - PRIMARY AND SECONDAR PREVENTION 05.00 - RISK FACTORS AND PREVENTION (PRIMARY AND SECONDARY) - 05.04 - STROKE COMPLICATIONS P2540 - ESOC25-1668 THE VALUE OF ELECTROENCEPHALOGRAPHY IN THE DETECTION OF DELAYED CEREBRAL ISCHEMIA FROM VASOSPASM: A LITERATURE REVIEW Athanasios Vasilopoulos 1 , Nikolaos Faraklas Perdikouris 2 , Athanasios Mitsias 2 , Ioannis Karakis 2,3 1 University Hospital of Lyon, Lyon, France, 2 School of Medicine, University of Crete, Heraklion Crete, Greece, 3 University Hospital of Heraklion, Heraklion Crete, Greece Background and Aims: Possible complications after subarachnoid hemorrhage (SAH) can be cerebral vasospasm and delayed cerebral ischemia (DCI), which majorly contribute to unfavourable neurological outcomes

On the neurological side, the pro-inflammatory factor released by AT may have first caused ferroptosis in the vagus nerve by inhibiting of the NRF2-GPX4 pathway, resulting in disorders of the autonomic nervous system
Incidence of chronic cystitis vs resolution at 28 dpi for nave, resolved and sensitized mice upon challenge infection
With sustained interdisciplinary collaboration, luteolin has the potential to evolve from an underrecognized nutraceutical into a rigorously validated therapeutic platform with meaningful impact on womens reproductive health and quality of life