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Moreover, the findings that mice with neutrophil-specific Gpx4 haploinsufficiency recapitulate key clinical features of human SLE, including autoantibodies, neutropenia, skin lesions and proteinuria, and that the treatment with a specific ferroptosis inhibitor significantly ameliorates disease severity in lupus-prone mice reveal the role of neutrophil ferroptosis in lupus pathogenesis

doi: 10.3389/fncel.2020.581950 Received 14 July 2020 Accepted 21 December 2020 Published 18 February 2021 Volume 14 - 2020 Edited by Danilo Bilches Medinas, University of Chile, Chile Reviewed by Jorge Matias-Guiu, Complutense University of Madrid, Spain

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