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The expression levels of Kim-1 and NGAL were increased 4 to 5-fold in the kidneys, resulting in significant nephrotoxicity

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Confusingly enough, cuprizone also does not kill oligodendrocytes in vitro [111], suggesting that cuprizone alone might not be enough to induce oligodendrocyte cell death in vivo

Advances in lipidomics, AI-driven receptor prediction, and structural biology are expected to accelerate the discovery of novel PRLMs and their receptors, while also providing deeper insights into their metabolic regulation [44]

HSYA suppressed ferroptosis in HUVEC, increased GSH, SLC7A11, and GPX4 levels, and significantly reduced atherosclerotic plaque formation