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Together, our data suggest that p53 controls a stress-responsive gene programme, rather than a single downstream gene, that facilitates fasting-dependent recruitment of macrophages into eWAT under metabolic stress

Chang Gung Med J 35 , 211218 (2012)

Hepatology 30(1):186195 Win S et al (2021) Hepatic mitochondrial SAB deletion or knockdown alleviates diet-induced metabolic syndrome, steatohepatitis, and hepatic fibrosis

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However, the effect was present only when caloric intake was not strictly controlled