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Exploring the relationship between urinary phthalate metabolites and Crohns disease via oxidative stress, and the potential moderating role of gut microbiota: A conditional mediation model

Additional studies are needed to evaluate how the performance of AFP as a screening test for HCC varies by etiology of liver disease and whether the threshold value that determines a positive screening test needs to vary by etiology of liver disease

Altogether, these complex triangular interactions between viral components, innate immunity and hepatocyte metabolism may explain why chronic hepatitis infections progressively lead to liver inflammation and progression to cirrhosis, fibrosis and hepatocellular carcinoma (HCC)

Most published data comes from in vitro and animal studies

In the mixed glial cells of ABCD1-knock-out mice, loss of AMPK induced mitochondrial dysfunction caused by reduced PGC-1 levels and spontaneous increased pro-inflammatory tendencies (111, 112)