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J Biol Chem 2012

Autonomic dysreflexia in spinal cord injury

DNA repair is not involved in the cell resistance To investigate whether U138MG resistance to cisplatin was because of an enhanced DNA repair capacity, host-cell reactivation (HCR) assays were performed using a firefly luciferase gene reporter

Figure 4B, C shows a clear and significant difference in weight-adjusted vascular resistance between kidneys from donors with a history of hypertension and those without, and those with histopathological findings of donor vascular disease (arterial fibroelastosis) compared to those without

For this purpose, we will summarize: 1) The current evidence that acutely-induced oxidative stress is cholestatic, by describing the mechanisms underlying the hepatocyte secretory failure, including the disorganization of the actin cytoskeleton and its most noticeable consequences, the impairment of tight-junctional structures and the endocytic internalization of canalicular transporters relevant to bile formation