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GHK-Cu stimulates angiogenesis primarily through upregulation of key growth factors including vascular endothelial growth factor (VEGF) and basic fibroblast growth factor (bFGF/FGF-2), with research demonstrating that GHK-Cu at nanomolar concentrations increases VEGF expression in dermal fibroblasts and stimulates human umbilical vein endothelial cell (HUVEC) proliferation through enhanced VEGF and FGF-2 expression

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Elevated levels of NNMT activity is associated with reduced cellular energy expenditure and increased fat storage

By activating AMPK pathways , MOTS-c boosts energy production, improves glucose uptake, and promotes fat oxidation

This relationship is stronger in women than in men (den Heijer et al, 1996)