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Hou et al., identified HDAC5 is a potential downstream target of KRAS via an iKPC mice model

9 Controversies in astrocyte-neuron metabolic coupling: the lactate shuttle and alternative viewpoints A significant and ongoing debate in brain energy metabolism centers on the quantitative importance and primary pathway of fuel delivery to activated neurons

A possible role for mitochondrial dysfunction in migraine

Bersuker K, Hendricks JM, Li Z, Magtanong L, Ford B, Tang PH, et al

Mutant KRAS increases the expression of glucose transporter 1 (GLUT1) and rate-limiting glycolytic enzymes, including hexokinases, phosphofructokinase 1 (PFK1), and lactate dehydrogenase A (LDHA), promoting glycolytic activity and increasing lactate production [17, 18]