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Application of CYP102A1M11H as a tool for the generation of protein adducts of reactive drug metabolites

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This reduction may influence extracellular matrix remodeling, immune cell recruitment, and paracrine signaling in surrounding tissues

GrK does not cleave off membrane-bound VEGFR1 from the cell surface, does not release potential sVEGFR1 storage pools from endothelial cells, and does not trigger sVEGFR1 release via protease activating receptor-1 (PAR-1) activation