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Inhibition of miR4943p alleviates oxidative stressinduced cell senescence and inflammation in the primary epithelial cells of COPD patients

This microorganism is responsible for a broad spectrum of diseases, ranging from mild infections such as otitis media and sinusitis to severe conditions including meningitis, pneumonia, and bacteremia [1]

Given the drugs success in tumor penetration, the researchers investigated whether adding immune checkpoint inhibitors to treatment with the drug would be even more effective, taking the brakes off T cells in more than one way at the same time but there was no such improvement

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