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Friedreich ataxia pathogenesis and implications for therapies

Moreover, our group recently demonstrated that IGF-1 gene therapy resulted in significant changes in several parameters related to microglia and astrocyte phenotypes (particularly in the CPu and dorsal hippocampal areas), supporting the use of IGF-1 as a therapeutic molecule for neuroinflammatory processes [87]

doi: 10.1038/s41573-020-00135-8

In addition, butylmalonate, an inhibitor of DIC, significantly decreased mGSH, suggesting DIC as the major GSH transporter in mouse cerebral cortical mitochondria (Kamga et al., 2010)

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