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& Fagard, R

Arnold) containing a C-terminal HRV-3C protease cleavage site (3C-tag: LEVLFQGP) followed by a GSG linker and 8His tag

RGC degradation is followed by a neuroinflammatory scar response via glial activity by expressing major histocompatibility complex class II molecules and components of the complement cascade that could exacerbate the pathogenic conditions and further promote RGC loss in POAG [24,25]

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