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On the contrary, increased expression of miR-155 facilitated the expansion and prolonged presence of Ex T cells [55]

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Peptidoglycan biosynthesis, lysine biosynthesis, and sulfur metabolism were not regulated by XJZ-C after subtracting the complement drug from XJZ, suggesting that the complement drug played a major role in the regulation of the above 3 metabolic pathways by XJZ

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