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Endothelial TLR4 activation impairs intestinal microcirculatory perfusion in necrotizing enterocolitis via eNOS-NO-nitrite signaling

Our data are consistent with the notion that energy substrates and glutathione produced in the liver are shuttled systemically and to the kidney to meet the metabolic demands of kidney repair and maintenance of redox balance

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For example, in the study of traumatic brain injury in mice, the lack of GPX1 increased the attack of ROS on hippocampal neurons, causing damage to post-traumatic memory function ( Interestingly, we found in our study that GPX1 is not capable of oxidative stress in all cases

10.1016/j.molcel.2012.10.007 74 WongC