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The increase in AA-induced by various factors and the activation of inflammatory cells promote ferroptosis by impairing mitochondrial function and generating large amounts of free radicals

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Hereby, lipid composition in VLDL provides a unique window to assess hepatocellular lipid homeostasis

Additionally, increasing studies have revealed the important mechanisms in the communication of mitochondria with other parts of cells (16), including cytochrome c release to induce cell death, 5-adenosine monophosphate-activated protein kinase activation to control mitochondrial fission and fusion, ROS generation to activate transcription factors, and mtDNA release to activate immune responses (Figure 1)

Dietary intake and circulating concentrations of carotenoids and risk of type 2 diabetes: a doseresponse meta-analysis of prospective observational studies