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So chlorpromazine, originally a drug for the dopamine receptor, happens to bind some other target, maybe dynamins, with much weaker affinity, and is thus a micromolar inhibitor of endocytosis

Consequently, a crucial task will be to select the more active agents and integrate them effectively in well-designed clinical trials incorporating predictive biomarkers such as gene mutations, amplifications, and immunohistochemical c-MET overexpression

Even tho i cannot 100% say for sure that BPC-157 was the reason that both treated areas are now feeling better after the therapy I can with certainty say that i did not experience any systemic healing effects on untreated areas that are now exhibiting similar symptoms

Chambers I, Silva J, Colby D, Nichols J, Nijmeijer B, Robertson M et al

Yet the same companies often provide reconstitution calculators, dosing protocols, and link to forums discussing human use protocols