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Indeed, mitochondria can produce both NO, by the activity of a Ca 2+ -sensitive mitochondrial NOS (mtNOS) (140, 528), and superoxide, following the partial reduction of oxygen within the mitochondrial matrix due to the natural leak of electron from the respiratory chain

Melatonin attenuates the postischemic increase in blood-brain barrier permeability and decreases hemorrhagic transformation of tissue-plasminogen activator therapy following ischemic stroke in mice

Future well-designed trials are needed to address these questions, as well as to identify the most suitable assessment tools for both research and clinical applications