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Thus, NPD4928 stabilizes endogenous FSP1 in a dose-dependent manner, indicating that NPD4928-dependent ferroptosis is primarily mediated through the inhibition of FSP1 activity

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Insulin also significantly increased the MR effect on CHAC1 and SLC7A5 expression but had no effect on other ATF4-responsive genes shown to be sensitive to mTOR (e.g., PSAT1, ASNS, CARS, MARS, SESN2, EIF4EBP1, and DDIT4 (data not shown))

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Carbamidomethylation of cysteines was defined as a fixed modification, while protein N-terminal acetylation and methionine oxidation were defined as variable modifications for database searching