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Aging is usually accompanied by dysregulation of the TSP, and increased flux via the TSP is a key target to modulate cognitive function, slow the aging process, and increase lifespan 8,10

Activation of c-Jun-N-terminal kinase is required for apoptosis triggered by glutathione disulfide in neuroblastoma cells

Liposomal glutathione formulations bypass this issue by encapsulating the molecule in lipid vesicles that survive gastric acid and are absorbed intact in the small intestine