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Researchers have proposed that the small tripeptide may use the PEPT2 transporter to cross intestinal and blood-brain barriers, but exact human bioavailability has not been precisely quantified

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Levels of the cardiomyocyte hypertrophy marker BNP were significantly increased in PE-treated cells, while PQQ treatment failed to fully reverse this change in si-YAP transfected cardiomyocytes (Figure 5D)