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Statements Acknowledgments This work was supported in part by a grant-in-aid for research from the Ministry of Education, Culture, Sports, Science and Technology (MEXT) of Japan (24390094

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Abstract Rationale Glutathione (GSH) is a key scavenger for cellular free radicals, and patients with bipolar disorder may have lowered GSH levels in plasma and in post-mortem brain tissue

(2021) described that Hcy activates the epithelial sodium channel and consequently induces endothelial dysfunction via reactive oxygen species (ROS)/COX-2-dependent activation of SGK-1/Nedd4-2 signaling [85]

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