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In myocardial ischemia, hypoxia and reoxygenation trigger an increase in free radical production in cardiac tissue (Elahi et al., 2009)

This is because they need to overcompensate for the extremely poor absorption rate of synthetic B12, not to mention the stress it puts on our body trying to convert that much synthetic B12 into usable B12

Not only that, 12/15-LOX inhibitors could improve neuronal damage and reduce edema and infarct size

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