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Collectively, these findings demonstrate that bacterial-induced mitochondrial dysfunction serves as a central signaling hub that coordinates innate immune activation and cell death programs

Karabowicz et al

Lncrnas56464.1 as a ceRNA Promotes the Proliferation of FibroblastLike Synoviocytes in Experimental Arthritis via the Wnt Signaling Pathway and Sponges Mir1523p