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Exendin-4, as a GLP-1R agonist, inhibits ERK1/2 by activating the PI3K/AKT signaling pathway

Notably, more than 95% of Kupffer cells expressed TIM4, a marker of embryonically-derived Kupffer cells 25 , indicating that repopulation by monocyte-derived macrophages (which are typically TIM4 ) was minimal under these conditions (Supplementary Fig