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Third, dopaminergic neurons have increased vulnerability to oxidative stress because they produce a large amount of dopamine, which can be oxidized by ROS, subsequently resulting in lysosomal dysfunction and -synuclein accumulation 9

Although Ucp2 knockout mice showed a higher level of cellular ATP production and enhanced GSIS, [86] which tends to support the above idea, two independent studies reported that Ucp2 -ablated fully back-crossed mice failed to show improved GSIS [87, 88]

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