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In probing the mechanisms of ferroptosis within the hepatic microenvironment, we question whether such mitochondrial anomalies are a driving force or simply a corollary of ferroptosis

doi: 10.3390/biom14020198 17

The complex structure was determined by molecular replacement (MR) with PHASER 63 using the crystal structures of the CHIKV prefusion E1E2 heterodimer (PDB ID 6JO8 28 ) and Fab (Alphafold2 generated) as search models

Rational drug design, which is the application of a structure-function relationship, is now widely used in modern medicinal chemistry for developing exquisitely selective ligands