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To determine the point at which mitochondrial Fe-S protein function is compromised by cystine starvation, we assessed respiratory complex activity in cells subject to cystine deprivation for 096 h that were concomitantly treated with ferrostatin-1 to prevent the induction of ferroptosis

This pathway is the fastest means of ATP production, but like ATP, creatine phosphate stores are quickly depleted

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In the course of these studies, we unexpectedly observed that these agents upregulate frataxin protein in frataxin-competent clonal rat cells (Supplemental Figure 2)

J Agric Food Chem 53(9):34213425