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In contrast, LCACs are thought to have the potential to activate inflammation, activating the NF-B luciferase reporter gene in RAW 264.7 cells in an acyl chain length- and concentration-dependent manner, as well as the phosphorylation of JNK and ERK [54, 55]

The transcription factor Myc controls metabolic reprogramming upon T lymphocyte activation

Second, the cross-sectional design limits our ability to capture dynamic changes in metabolism over the course of disease progression or treatment beyond the two-week window analyzed in the T1D cohort

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