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Multivariate analysis showed that the GSTM1-null genotype significantly correlated with the presence of (moderatesevere) SOS ( P =0.026)

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These genomic alterations enable Leishmania to develop resistance through various mechanisms including altered drug transport, target modification, and enhanced metabolic detoxification pathways (Ponte-Sucre et al., 2017)

In summary, although current studies suggest that ferroptosis plays a central regulatory role in the pathological sequence of PVLlinking hypoxiaischemia, inflammatory infiltration, and white-matter necrosisand provides a new mechanistic basis for understanding the pathway from periventricular iron accumulation to oligodendrocyte loss and motor developmental impairment, several essential gaps remain

5, Additional file 1: Figure S5)